Items where Author is "Nie, Yinyue"
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apoptosis
Nie, Yinyue, Huang, Luyue ORCID: 0000-0001-5214-2973 , Wang, Luchuan, Zeng, Pei, Gong, Chao ORCID: 0000-0001-5214-2973 , Song, Lin, Guo, Jin and Zhou, Shaobo ORCID: 0000-0001-5214-2973 (2024) Vitamin D3 treatment reduced epileptic neuronal damage by inhibiting apoptosis and increasing vitamin D receptor expression in an in vivo epileptic model. Journal of Integrative Neuroscience. ISSN 0219-6352 (Print), 1757-448X (Online) (In Press)
epilepsy
Nie, Yinyue, Huang, Luyue ORCID: 0000-0001-5214-2973 , Wang, Luchuan, Zeng, Pei, Gong, Chao ORCID: 0000-0001-5214-2973 , Song, Lin, Guo, Jin and Zhou, Shaobo ORCID: 0000-0001-5214-2973 (2024) Vitamin D3 treatment reduced epileptic neuronal damage by inhibiting apoptosis and increasing vitamin D receptor expression in an in vivo epileptic model. Journal of Integrative Neuroscience. ISSN 0219-6352 (Print), 1757-448X (Online) (In Press)
rats
Nie, Yinyue, Huang, Luyue ORCID: 0000-0001-5214-2973 , Wang, Luchuan, Zeng, Pei, Gong, Chao ORCID: 0000-0001-5214-2973 , Song, Lin, Guo, Jin and Zhou, Shaobo ORCID: 0000-0001-5214-2973 (2024) Vitamin D3 treatment reduced epileptic neuronal damage by inhibiting apoptosis and increasing vitamin D receptor expression in an in vivo epileptic model. Journal of Integrative Neuroscience. ISSN 0219-6352 (Print), 1757-448X (Online) (In Press)
vitamin D receptor
Nie, Yinyue, Huang, Luyue ORCID: 0000-0001-5214-2973 , Wang, Luchuan, Zeng, Pei, Gong, Chao ORCID: 0000-0001-5214-2973 , Song, Lin, Guo, Jin and Zhou, Shaobo ORCID: 0000-0001-5214-2973 (2024) Vitamin D3 treatment reduced epileptic neuronal damage by inhibiting apoptosis and increasing vitamin D receptor expression in an in vivo epileptic model. Journal of Integrative Neuroscience. ISSN 0219-6352 (Print), 1757-448X (Online) (In Press)
vitamin D3
Nie, Yinyue, Huang, Luyue ORCID: 0000-0001-5214-2973 , Wang, Luchuan, Zeng, Pei, Gong, Chao ORCID: 0000-0001-5214-2973 , Song, Lin, Guo, Jin and Zhou, Shaobo ORCID: 0000-0001-5214-2973 (2024) Vitamin D3 treatment reduced epileptic neuronal damage by inhibiting apoptosis and increasing vitamin D receptor expression in an in vivo epileptic model. Journal of Integrative Neuroscience. ISSN 0219-6352 (Print), 1757-448X (Online) (In Press)